Semeiotica Biofisica Quantistica. Il Nuovo Rinascimento della Medicina. www.sisbq.org

Articoli con tag ‘Inherited Real Risks’

Vallebona’s Manoeuvre*. Under renal experimental decongestion, the significant increase in blood flow in oncological tissue plays a central diagnostic and differential diagnostic role.

“Vivi le domande ora. Forse poi, in qualche giorno lontano nel futuro, inizierai gradualmente, senza neppure accorgertene, a vivere a tuo modo nella risposta.”

RAINER MARIA RILKE

As shows Oncological Terrain-dependent, Inherited  Real Risk,  tumour microcirculation differs from that of normal tissues, from Cllinical Microangiology point of view (1-4).

Authors observed that elevation of blood pressure produced a several-fold increase in tumour blood flow without increasing blood flow in normal tissue(5). These results indicate that the delivery of systemically administered anticancer drugs could be selectively enhanced in tumour tissues by induced hypertension. Unfortunately,  no one has ever thought to use the different microcirculatory behaviour in cancer for diagnostic and differential diagnostic purposes (6-10).

Recent advances have improved our understanding of the renin-angiotensin system (RAS). These have included the recognition that angiotensin (Ang 1-7) is a biologically active product of the RAS cascade. The identification of the ACE homologue ACE2, which forms Ang-1-7) from Ang II, and the GPCR Mas as an Ang-1-7 receptor have provided the necessary biochemical and molecular background and tools to study the biological significance of Ang-1-7 (5-7).

Most available evidence supports a counter-regulatory role for Ang-1-7 by opposing many actions of Ang II on AT₁ receptors, especially vasoconstriction. Many studies have now shown that Ang-1-7 by acting via Mas receptor exerts inhibitory effects on inflammation and on vascular and cellular growth mechanisms.

The homogeneous  data of my ongoing research, observed on 23 individuals of both sexes, aged from 12 to 44 years, taken together, may help in paving the original way, microcirculatory in nature, for the development of novel diagnostic and differential diagnostic  procedure for cancer, starting from its initial stage of Oncological Terrain-dependent, Inherited Real Risk.

In previous artiches,  I have developed the clinical evaluation of the RAS, demonstrating the real reliability of Quantum-Biophysical-Semeiotics in both diagnostic and researc (13).

Interestingly, due to the presence of no local realm in all biological systems, in one second doctors, skilled in  Quantum-Biophysical-Semeiotics , may bedside recognize any disorders in urinary tract by means of Pollio’s Sign (13, 14).

In healthy, intense (1.000 dyne/cm.2) cutaneous pinching of kidney trigger-points does not bring about simultaneously the Gastric Aspecific Reflex (Fig- 1).

Fig.1

Gastro-Aspecific Reflex. In the stomach, Both fundus and body are dilated , while antral-pyloric region contracts.

 

On the contrary, in the presence of any disease of the urinary tract, such a reflex appears simultaneously: positive Pollio’ Sign.

Soon thereafter physicians can localize the precise site of disorder, ascertaining its real nature.

Interestingly, as regards early diagnosis of renal artery stenosis, Quantum Biophysical Semeiotics allows doctor to bedside recognize this vascular disorder of the kidney, since its initial stage of Inherited  Real Risk.

Perhaps, for instance, available evidence does not clearly support one treatment approach over another for atherosclerotic renal artery stenosis. However, we must admit that patients with such a disorder are properly diagnosed exclusively a long time after initial disease onset, as in our case.

Unfortunately, all around the world, General Practitioners know only the traditional physical semeiotics, that isn’t so efficacious to allow doctor to recognize, since its first stage, Renal Artery Stenosis. Nowadays, physicians are capable to bedside recognize  from birth any real risk of kidney diseases, both oncological and degenerative in nature.

In order to recognize Renal Artery Stenosis, the following easy and quick manoeuvre proved to be really effective in my long year clinical experience.

In health, doctor first of all delimits kidney area (15). Soon therafter doctor increases the pressure of stethoscope bell-piece, localized on kidney cutaneous projection area, causing kidney dilation (due to its congestion) immediately followed by kidney size reduction (due to decongestion) to its minimal value.

At this point, pressure prompt interruption causes the  rapid – in 2 sec or less – return of kidney to its normal, basal size, indicating a physiological blood flow in renal artery.

On the contrary, in case of renal artery stenosis,  the  latency time results more than 2 sec., in relation to the severity of underlying disease.

To stimulate RAS, physician stimulates any one renal trigger point by cutaneous intense pintching, that causes  renal decongestion and secretion of renin. In reality,  this procedure is simultaneously followed by physiological Microcirculatory Activation, type I, associated (1, 2)  in both the adrenal gland  (catecholamines) and in the liver (angiotensinogen).

In whatever normal tissue, after the intense stimulation of kidney trigger points, the Latency Time of Gastric Aspecific Reflex slowly lowers. For instance, basal Liver- GA Reflex is  8 sec., and after the renal decongestion lowers to about 7 sec.

On the contrary, in the cancer, even in its real initial stage of Inherited Real Risk, latency Time of gastric aspecific reflex increases, and its augmentation is related directly to the stage, i.e. to the seriousness of disorder.

Due to obvious reasons, I do not illustrate the refined and reliable signs of Clinical Microangiology (1, 2), namely the fascinating behaviour of the peripheral heart, according to Claudio Allegra,  under Vallebona’s Manoeuvre. Interestingly, these microcirculatory events represent the “Implicate Order” (D. Bohn), which underlies the clinical QBS phenomenology, illustrated above.

Importantly, the provisional but homogeneous data of an  ongoing research, started recently, show the identical behaviour of chronic degenerative non-oncological tissues, as CVD, Osteoporosis, and T2DM.

If these data will be corroborated on a large scale by other Authors, the Vallebona’s Manoeuvre may help in paving another  original way in  clinical diagnostics.

* Manoeuvre dedicated to my unforgettable friend Enrico Vallebona,

References.

  1. Sergio Stagnaro. Introduzione alla Microangiologia Clinica 10 dicembre 2011. www.sisbq.org, http://www.sisbq.org/uploads/5/6/8/7/5687930/mc_intro.pdf
  2. Sergio Stagnaro – Marina Neri Stagnaro. Microangiologia Clinica. A cura di Simone Caramel. e-book, http://www.sisbq.org, http://www.sisbq.org/uploads/5/6/8/7/5687930/microangiologiaclinicasbq2016.pdf.
  3. Stagnaro Sergio.Reale Rischio Semeioticso Biofisico. I Dispositivi Endoarteriolari di Blocco neoformati, patologici, tipo I, sottotipo a) oncologico, e b) aspecifico. Ediz. Travel Factory, www.travelfactory.it, Roma, 2009.
  4. Sergio Stagnaro and Simone Caramel. BRCA-1 and BRCA-2 mutation bedside detection and breast cancer clinical primary prevention.  Front. Genet. | doi: 10.3389/fgene.2013.00039.  http://www.frontiersin.org/Cancer_Genetics/10.3389/fgene.2013.00039/full [MEDLINE]
  5. Suzuki M, Hori K, Saito S, Tanda S, Abe I, Sato H, Sato H. Functional characteristics of tumour vessels: selective increase in tumour blood flow. Sci Rep Res Inst Tohoku Univ Med. 1989 Dec;36(1-4):37-45.
  6. Robson Augusto Santos. Angiotensin-(1–7). Hypertension. 2014;63:1138-1147
  7. Simões e Silva AC, Silveira KD, Ferreira AJ, Teixeira MM. ACE2, angiotensin-(1-7) and Mas receptor axis in inflammation and fibrosis. Br J Pharmacol. 2013 Jun;169(3):477-92. doi: 10.1111/bph.12159.
  8. Matthew J. Durand, Natalya S. Zinkevich, Michael Riedel, et al. Vascular Actions of Angiotensin 1–7 in the Human Microcirculation – Novel Role for Telomerase. Arterioscler Thromb Vasc Biol. 2016 Jun; 36(6): 1254–1262.
  9. Suuzuki M, Abe I, Hori K, Saito S, Sato H: Characteristic blood circulation in tumour tissue, with reference to local permeation of drug in cancer chemotherapy. In: Proceedings of the 36th annual meeting of the Japanese Cancer Association, Tokyo. Tokyo: Japanese Cancer Association, 1977, p 149.
  10. Suzuki M, Hori K, Abe I, Saito H: Characteristics of microcirculation in tumour. Jpn J Cancer Chemother 6 (Suppl II): 287–291, 1979.
  11. Stagnaro-Neri M, Stagnaro S., Valutazione clinica percusso-ascoltatoria del sistema nervoso vegetativo e del sistema renina-angiotensina, circolatorio e tessutale. Arch. Med. Int. XLIV, 17378, 1992.
  12. Stagnaro Sergio.   Renal Artery Stenosis: bedside rapid Diagnosis even in its initial stage with Quantum-Biophysical-Semeioticss. 23 May 2009 http://sciphu.com/, http://sciphu.com/2009/05/renal-artery-stenosis-bedside-rapid.html  and   http://wwwshiphusemeioticsscom-stagnaro.blogspot.com/
  13. Stagnaro Sergio e Paolo Manzelli. L’Esperimento di Lory. Scienza e Conoscenza, N° 23, 13 Marzo 2008. http://www.scienzaeconoscenza.it//articolo.php?id=17775
  14. Sergio Stagnaro. Realtà Locale e Non-Locale nella Medicina del Nuovo Rinascimento. https://dabpensiero.wordpress.com/2016/06/03/realta-locale-e-non-locale-nella-medicina-del-nuovo-rinascimento/
  15. Stagnaro-Neri M., Stagnaro S. Introduzione alla Semeioticsa Biofisica. Il Terreno Oncologico. Travel Factory, Roma, 2004. http://www.travelfactory.it/semeioticsa_biofisica.htm
Annunci

Quantum-Biophysical-Semeiotic bedside Diagnosis of Frontal-Temporal Dementia, starting from its Inherited Real Risk.

Frontal-temporal dementia is a form of dementia that occurs when the frontal-temporal lobes of the brain are damaged genetically through mother impaired mitochondria. Experts estimate that it is responsible for 10%-15% of dementia cases (1-17).

Frontal-temporal dementia, based on its Inherited Real Risk I have recently discovered, has its own constellation of symptoms and is separate from Alzheimer’s disease, because in AD typically there is not cerebral physiological Microcirculatory Activation, associated, type I, under the Insulinemic Acute Pick Test (18-20).

In addition, but less important from the quantum biophysical semeiotic differential diagnosis point of view, one difference between frontal-temporal dementia (FTD) and Alzheimer’s disease is that, on average, frontal lobe dementia firstly presents itself significantly earlier in life. FTD symptoms usually appear between 45 and 65 years of age, while the majority of Alzheimer’s cases occur in those over 65.

The frontal lobes are responsible for helping inhibition and behavior regulation, so people with frontal lobe dementia will often exhibit strange or unusual behaviors and personality changes. In fact, personality changes and behavior problems are hallmarks of the disorder, whose synptoms appear 4-5 decades after birth, if physician ignores the Inherited Real Risk of FTD, present at birth, diagnosed with a stethoscope and removed by inexpensive therapy (1-15).

Personality changes due to FTD include: Impulsiveness, Apathy and indifference, socially inappropriate behavior

People with frontal lobe dementia may suddenly struggle with binge eating and gambling compulsions because of the impulsiveness associated with frontal lobe atrophy

Frontal-temporal dementia does not cause memory loss, but it can other cognitive and neurological problems similar to those caused by Alzheimer’s disease or stroke.

These symptoms can include: Difficulty with speech and language; Inability to concentrate, Inability to pla; Using the wrong object for the wrong task, or at the wrong time; Movement and balance difficulties.

However, nowadays physicians can perform differential diagnosis at the bedside clearly an quickly by means of characteristic quantum biophysical semeiotic signs (20, 21).

Fronto-temporal dementia and parkinsonism (FTDP) is a major neurodegenerative syndrome, particularly for those with symptoms beginning before age 65. This variant of FTD is characterized by the association of PD signs, especially the data of prolactin test (= the breast massage causes a gastric aspecific reflex, more persistent of 7 sec., physiological value) and the impairment also of the posterior third of parietal lobe, typically undamaged in FTD. (See later on)

Although Inherited Real Risk is always present in FTD, a spectrum of degenerative disorders since ever notoriously present as sporadic or familial FTD.

Mutations in the gene encoding the microtubule associated protein tau (MAPT) on chromosome 17 have been found in many kindreds with familial FTDP. Several other kindreds with FTDP had been linked to chromosome 17, but they had ubiquitin-positive inclusions rather than tauopathy pathology, and no mutations in MAPT. This conundrum was solved over this past year with the identification of mutations in the gene encoding progranulin (PGRN), which is only 1.7 Mb centromeric to MAPT on chromosome 17. Authors have compared and contrasted the demographic, clinical, radiologic, neuropathologic, genetic, and pathophysiologic features in patients with FTDP linked to mutations in MAPT and PGRN, highlighting the many similarities but also a few important differences (16, 17).

There is a general agreement that no one test is able to diagnose Frontal-temporal Dementia. Instead physicians are able to use the balance of evidence to diagnose frontal-temporal dementia based on their best judgment. Because there is no foolproof test and diagnosis depends on the physician’s knowledge, judgment, and observation of the patient, FTD is notoriously difficult to diagnose in its early stages, if physician ignore Quantum Biophysical Semeiotic. Although as the disease progresses, it becomes easier to definitely distinguish it between other disorders (15-18).

Behavioral changes are the most common early signs of the variant GRN-related frontotemporal dementia. These include marked changes in personality, judgment, and insight. It may become difficult for affected individuals to interact with others in a socially appropriate manner. Affected people may also become easily distracted and unable to complete tasks. They increasingly require help with personal care and other activities of daily living.

Many people with GRN-related frontotemporal dementia develop progressive problems with speech and language (FTDP). Affected individuals may have trouble speaking, remembering words and names, and understanding speech. Over time, they may completely lose the ability to communicate.

As all other numerous Inherited Real Risks, Fronto-temporal  Dementia heritable Risk does really exist, bedside diagnosed from birth, and treated with Reconstructing Mitochondrial Quantum Therapy (20-25). Twenty years ago, I have discovered Constitution-Dependent, Inherited Real Risks of chronic degenerative disorders, as CVD, T2DM, Osteoporosis, Cancer and of a flurry of neurodegenerative disorders: www.semeioticabiofisica.it.

All mothers of patients affected by FTD are positive to their own Inherited Real Risk.

Recently I’ve discovered the Inherited Real Risk of Fronto-temporal Dementia, that I predict is also eliminated with inexpensive Reconstructive Mitochondrial Quantum Therapy, as  all other risks, mentioned above, dependent on mitochondrial cytopathy (CAEMH, Congenital Acidosic Enzyme-Metabolic Histangiopathy) transmitted through the mother (22-27).

The diagnostic procedure aiming to bedside recognize such a risk, even in apparently normal individuals, starting from the birth, begins with the evaluation of  Oculo-gastric aspecific reflex: in a subjects, lying down in supine position with closed eye, physician  presses with moderate intensity (500 dyne / cm2) with a fingertip on the eyeball on one side and then the other (28).

In healthy, after a Latency Time of 8 sec., in the stomach appears dilation of both fundus and body, while antral redgion contracts, Gastric Aspecific Reflex. Reflex intensity is less than 1 cm., and the Duration < 3 sec. – 4 sec. <

On the contrary, in subject involved by FTD Inherited Real Risk, the Reflex Parametric Values, i.e., Latency Time, Reflex Duration and Intensity, are pathologically modified, in relation to the seriousness of underlying disorder (28).
Soon thereafter, the physician assesses the Cerebro-gastric aspecific Reflex  by pressing with medium intensity pressure by means of a fingertip (700 dyne / cm.2) on the skin projection area of brain frontal, parietal, temporal, occipital, and cerebellar lobes.

In FTD such a reflex shows pathological parameter values, i.e., Latency Time (NN = 8 sec.), reflex Duration (NN < 3 sec. – 4 sec.<) and intensity (NN less tham 1 cm.), when are stimulated the trigger points of frontal lobe, anterior and middle temporal, and cerebellar lobes.

Interestingly, in FTD the posterior third of temporal lobe is normal: differential diagnosis. Exclusively in the FTDP also parietal lobes are pathologically modified

As in all forms of dementia, also in the FTD the Cerebellar-Gastric Aspecific Reflex shows patological parametric values starting from birth, because the cerebellum is a sensor of dementia (29).

As it is known, above-referred parametric values are related to the severity of the underlying disease. Therefore, the described clinical evaluation plays a central role in therapeutic monitoring of Frontal-temporal Dementia under treatment with Reconstructing Mitochondrial Quantum Therapy (20, 24, 27).

References

  1. Bang J, Spina S, Miller BL. Frontotemporal dementia. Lancet 2015; 386: 1672–1682. [Medline]
  2. Kurz A, Kurz C, Ellis K, et al. What is frontotemporal dementia?Maturitas 2014; 79: 216–219. [Medline]
  3. Bott NT, Radke A, Stephens ML, et al. Frontotemporal dementia: diagnosis, deficits and management. Neurodegener Dis Manag 2014; 4: 439–454. [Medline]
  4. World Health Organization. Dementia fact sheet, http://www.who.int/mediacentre/factsheets/fs362/en/ (2016). Accessed January 22, 2017.
  5. Rosso SM, Donker Kaat L, Baks T, et al. Frontotemporal dementia in The Netherlands: patient characteristics and prevalence estimates from a population-based study. Brain 2003; 126: 2016–2022. [Medline]
  6. Onyike CU, Diehl-Schmid J. The epidemiology of frontotemporal dementia. Int Rev Psychiatry 2013; 25: 130–137. [Medline]
  7. Knopman DS, Roberts RO. Estimating the number of persons with frontotemporal lobar degeneration in the US population. J Mol Neurosci 2011; 45: 330–335. [Medline]
  8. Lambert MA, Bickel H, Prince M, et al. Estimating the burden of early onset dementia; systematic review of disease prevalence. Eur J Neurol 2014; 21: 563–569. [Medline]
  9. Rascovsky K, Hodges JR, Knopman D, et al. Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia. Brain 2011; 134: 2456–2477. [Medline]
  10. Gorno-Tempini ML, Hillis AE, Weintraub S, et al. Classification of primary progressive aphasia and its variants. Neurology 2011; 76: 1006–1014. [Medline]
  11. Leyton CE, Hodges JR. Towards a clearer definition of logopenic progressive aphasia. Curr Neurol Neurosci Rep 2013; 13: 396. [Medline]
  12. Rohrer JD, Guerreiro R, Vandrovcova J, et al. The heritability and genetics of frontotemporal lobar degeneration. Neurology 2009; 73: 1451–1456. [Medline]
  13. Rademakers R, Neumann M, Mackenzie IR. Advances in understanding the molecular basis of frontotemporal dementia. Nat Rev Neurol 2012; 8: 423–434.[Medline]
  14. Capozzo R, Sassi C, Hammer MB, et al. Clinical and genetic analyses of familial and sporadic frontotemporal dementia patients in Southern Italy. Alzheimers Dement 2017; 13: 858–869. [Medline]
  15. Moreno F, Indakoetxea B, Barandiaran M, et al. The unexpected co-occurrence of GRN and MAPT p.A152T in Basque families: clinical and pathological characteristics. PLoS One 2017; 12: e0178093. [Medline]
  16. Rademakers R, Cruts M, van Broeckhoven C. The role of tau (MAPT) in frontotemporal dementia and related tauopathies. Hum Mutat 2004; 24: 277–295. [Medline]
  17. Boeve BF, Hutton M. Refining frontotemporal dementia with parkinsonism linked to chromosome 17: introducing FTDP-17 (MAPT) and FTDP-17 (PGRN). Arch Neurol 2008; 65: 460–464. [Medline]

18) Stagnaro-Neri M., Stagnaro S., Semeiotica Biofisica: la manovra di Ferrero-Marigo nella diagnosi clinica della iperinsulinemia-insulino resistenza. Acta Med. Medit. 13, 125, 1997.

19) Sergio Stagnaro.      Manovra di Ferrero-Marigo e Vasomotilita’ a Riposo e Dopo Il Test Di Secrezione Del Picco Acuto Insulinemico nella Valutazione Clinica della Insulino Resistenza 23 novembre 2010.

http://qbsemeiotics.weebly.com/uploads/5/6/8/7/5687930/manovradiferrero.pdf

20) Marco Marchionni, Simone Caramel, Sergio Stagnaro. The Role of ‘Modified Mediterranean Diet’ and Quantum Therapy In Alzheimer’s Disease  Primary Prevention. Letter to the Editor, The Journal of Nutrition, Health & Aging, Volume 18, Number 1, 2014, Springer Ed. http://link.springer.com/article/10.1007/s12603-013-0435-7  [Medline]

21) Stagnaro S., Percussione Ascoltata degli Attacchi Ischemici Transitori. Ruolo dei Potenziali Cerebrali Evocati. Min. Med. 76, 1211, 1985 [Medline]

22) Sergio Stagnaro and Simone Caramel.  BRCA-1 and BRCA-2 mutation bedside detection and breast cancer clinical primary prevention.  Front. Genet. | doi: 10.3389/fgene.2013.00039.  http://www.frontiersin.org/Cancer_Genetics/10.3389/fgene.2013.00039/full [Medline]

23) Marco Marchionni, Simone Caramel, Sergio Stagnaro. Inherited Real Risk of Alzheimer’s Disease: bedside diagnosis and primary prevention.Frontiers in Neuroscience, in http://www.frontiersin.org/Aging_Neuroscience/10.3389/fnagi.2013.00013/full

24) Marco Marchionni, Simone Caramel, Sergio Stagnaro. The Role of ‘Modified Mediterranean Diet’ and Quantum Therapy In Alzheimer’s Disease  Primary Prevention. Letter to the Editor, The Journal of Nutrition, Health & Aging, Volume 18, Number 1, 2014, Springer Ed. http://link.springer.com/article/10.1007/s12603-013-0435-7  [Medline]

25) Sergio Stagnaro and Simone Caramel (2012). Quantum Therapy: A New Way in Osteoporosis Primary Prevention and Treatment. Journal of Pharmacy and Nutrition Sciences, (27 June 2012) | doi:10.1038/ejcn.2012.76, http://www.nature.com/doifinder/10.1038/ejcn.2012.76. PMID:22739250  [Medline]

26)) Stagnaro Sergio. Reale Rischio Semeiotico Biofisico. I Dispositivi Endoarteriolari di Blocco neoformati, patologici, tipo I, sottotipo a) oncologico, e b) aspecifico. Ediz. Travel Factory, www.travelfactory.it, Roma, 2009.

26) Caramel S., Marchionni M., Stagnaro S. Morinda citrifolia Plays a Central Role in the Primary Prevention of Mitochondrial-dependent Degenerative Disorders. Asian Pac J Cancer Prev. 2015;16(4):1675. http://www.ncbi.nlm.nih.gov/pubmed/25743850[MEDLINE]

27) Sergio Stagnaro and Simone Caramel (2013). The Role of Modified Mediterranean Diet and Quantum Therapy in Oncological Primary Prevention.  Bentham PG., Current Nutrition & Food Science  ISSN (Print): 1573-4013;  ISSN (Online): 2212-3881. VOLUME: 9,  ISSUE: 1; DOI: 10.2174/1573401311309010011;  http://www.benthamscience.com/contents-JCode-CNF-Vol-00000009-Iss-00000001.htm

28) Stagnaro-Neri M., Stagnaro S. Introduzione alla Semeiotica Biofisica. Il Terreno Oncologico. Travel Factory, Roma, 2004.   http://www.travelfactory.it/semeiotica_biofisica.htm

29) Sergio Stagnaro. Il Cervelletto è un Sensore della Predisposizione all’Aterosclerosi Cerebrale. La Manovra di De Lisi.  www.sisbq.org, http://www.sisbq.org/uploads/5/6/8/7/5687930/lisi_cervelletto_atscerebrale.pdf

Archives Internal Medicine conosce ed apprezza la Semeiotica Biofisica Quantistica, troppo difficile per i “Professori”.

Di seguito potete leggere un recente commento, inviato il 3 maggio 2012, accettato e messo in rete nel sito della rivista Arch. Int. Med., rivista dell’Associazione Medici Americani,  AMA, firmato  da me e dal Presidente della SISBQ www.sisbq.org , dott. Simone Caramel.

Anche uno scolaretto di Gregory Bateson e i “Professori”, forse,  possono comprenderne il senso ed il significato del commento.

Ho apprezzato sinceramente l’intelligenza, la lungimiranza e l’apertura mentale degli Editori della peer-review statunitense, qualità che finora non mi è stato possibile riconoscere nelle cosiddette competenti Autorità dell’assistenza sanitaria del mio Paese.

Con parole univoche e semplici, tali da essere comprese da chiunque, per l’ennesima volta affermo la mia convinzione sulle cause del silenzio delle competenti Autorità, dei “Professori” e dei Giornalisti italiani sulla Semeiotica Biofisica Quantistica.

Escluso il complotto, che considero una insostenibile e ridicola ipotesi, la causa del silenzio  è la incapacità dei nominati a comprendere ed  applicare la Semeiotica Biofisica Quantistica, di una difficoltà insormontabile a causa della loro dipendenza assoluta dal Laboratorio di Analisi Chimiche e dal Dipartimento delle Immagini.

Un “Professore”, pertanto, potrebbe considerare questo suo silenzio segno di onestà intellettuale. Infatti, non si dovrebbe criticare ciò  che non si conosce!

Non conosco la lingua araba e perciò non mi sento autorizzato a criticare le poesie d’amore di Nizar Qabbani, il poeta siriano che viene ritenuto uno tra i piu’ raffinati esponenti della poesia araba contemporanea:

O Signore, il mio cuore non mi basta piu’

quella che io amo e’ grande quanto il mondo:

mettimene nel petto un altro

che sia grande quanto il mondo.

Come si può immaginare un “Professore”,  sclerotizzato nel suo piccolo mondo medico, rassicurante e redditizio, studiare, nella pace del suo castello dal portone chiuso, la diagnosi semeiotico-biofisico-quantistica di ipotiroidismo subclinico, posta in un minuto con un fonendoscopio, mediante l’osservazione dell’attivazione microcircolatoria di tipo I, associata, nel centro neuronale del TSH-RH? Stagnaro Sergio.  Bed-Side Biophysical-Semeiotic Evaluation of Thyroid Dysfunction in Cardiology. Ann Int Medic. 21 May, 2008, http://annals.org/content/148/11/832.abstract/reply#annintmed_el_86312

Buona Lettura!

http://archinte.ama-assn.org/cgi/eletters/172/8/642#1180

Reducing NHS expense is possible with Physical Semeiotics Advances.

Sergio Stagnaro, Simone Caramel   (May 3, 2012)

Reducing NHS expense is possible with Physical Semeiotics Advances.

May 3, 2012

Sergio Stagnaro,

Simone Caramel,

Quantum Biophysical Semeiotics Research Laboratory,

Send reply to journal:
Re: Reducing NHS expense is possible with Physical Semeiotics Advances.

E-mail Sergio Stagnaro, et al.      In our opinion, based on a long clinical experience, NHS expense, including that in Emergency Department, can be reduced significantly if physicians would be told on the progress of physical semeiotics, e.g., Quantum Biophysical Semeiotics. This science is an extension of classical semiotics, a discipline of medicine that studies and interprets the signals of the human body in order to detect and diagnose diseases. The ‘Quantum Biophysical Semiotics’ develops according to a multidisciplinary approach that involves chemistry and biology, genetics and neuroscience, chaos theory and quantum physics. It is based on the method of auscultatory percussion, through which by simple means of the common stethoscope, it is possible to listen to the messages that the body gives us when appropriately stimulated. The stimuli, which can be percussion, pinching, and finger pressure of various intensities, are used to induce consistent behavior – typical of dissipative systems far from equilibrium as defined by Prigogine and comparable to the behavior of plasma studied by Bohm – in well-defined biological systems of the human body. Thus gives local qualitative information on the state of health or disease, whether potential, being developed but not yet evident by usual clinical trial, effective, or even in chronic phase.

‘Quantum Biophysical Semeiotics’ provides detailed case studies based on the duration, intensity, and latency time of the reflections, which are the central element of all the diagnostics, and on the basis of which it is possible to say that the presence of deterministic chaos, as measured by the fractal dimension, is an indicator of physiological state of the biological system investigated. This is always accompanied by a non-local reality, simultaneous and synchronic (as demonstrated at the sub-quantum by Aspect), parallel to the local one, where there is of course waste of energy in space-time. However, if the equilibria of type ‘chaotic or strange attractor’ give way to equlibria of type ‘limit cycle’ (periodic) or ‘fixed point’, this is a sign respectively of potential pathology and tendency to disease or to chronicity. The quantum aspect is reinforced by the fact that the reflections are not implemented in a continuous way, but are quantized and discontinuous, showing that constant feedback between implicit and explicit order, as suggested by Bohm. ‘Quantum Biophysical Semeiotics’ – QBS – can detect at birth the potential existence of well- defined diseases, such as cancer, diabetes mellitus, atherosclerosis, hypertension, ischemic heart disease, likely to be present only if maternal mitochondrial DNA is altered, which in turn leads to a particular mitochondrial cytopathy (painful condition of the cell) called CAEMH. In the case that cytopathy was intense, it gives rise from birth to specific QBS constitutions, grounds on which one can check the corresponding diseases, where there is evidence of their actual risk. For example, it can exist from birth the ‘Oncological Terrain’, which can lead to cancer Real Risk, which in turn allows at a certain point of the life of the onset of cancer. Another example is given by the arteriosclerotic constitution, which gives rise to the ‘Real Risk’ of coronary artery disease that may lead to unexpected death from myocardial infarction. Such may be the case with many young athletes, even if they are monitored cardiologically each year according to the traditional diagnosis. For further information, visit http://www.sisbq.org

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